This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Herpes Simplex Infections
Information about Herpes Simplex Infections: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Herpes Simplex Infections
Most recent studies have shown that Herpes Simplex Infections shares some biological mechanisms with congenital-herpes-simplex-infection, cytomegalovirus-infections, encephalitis, genital-herpes, herpes-labialis, herpes-zoster-disease, herpesviridae-infections, hiv-infections, immunologic-deficiency-syndromes, infective-disorder, keratitis, keratitis-dendritic, keratitis-herpetic, neonatal-herpes-simplex-virus-infection, nervousness, pregnancy-complications-infectious, recurrence-(disease-attribute), ulcer, virus-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Herpes Simplex Infections, and have been seen in publications frequently: Angiogenesis, Antibody-dependent Cellular Cytotoxicity, Cell Death, Cell Proliferation, Coagulation, Cytokine Production, Dna Replication, Excretion, Hypersensitivity, Immune Response, Inflammatory Response, Innate Immune Response, Localization, Lymphocyte Proliferation, Pathogenesis, Secretion, Translation, Transport, Viral Replication, Virulence
Quite a number of genes have been found to play important roles in Herpes Simplex Infections, such as CD4, CD8A, CSF2, IFNA1, IFNG, IL10, IL2, IL4, IL6, LAMC2, LAT, NFKB1, TG, TKT, TLR2, TNF, TNFRSF14. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.