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Disease Info Card
Hemoglobin F Disease
Information about Hemoglobin F Disease: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hemoglobin F Disease
Most recent studies have shown that Hemoglobin F Disease shares some biological mechanisms with acute-erythroblastic-leukemia, alpha-thalassemia, anemia, anemia-hemolytic, anemia-sickle-cell, beta-thalassemia, delta-beta-thalassemia, delta-thalassemia, diabetes-mellitus, fetomaternal-transfusion, glucosephosphate-dehydrogenase-deficiency, hematological-disease, hemoglobinopathies, hemolysis-(disorder), hemorrhage, hereditary-persistence-of-fetal-hemoglobin-thalassemia, pregnancy-complications-hematologic, sickle-cell-trait, thalassemia, thalassemia-intermedia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hemoglobin F Disease, and have been seen in publications frequently: Aging, Cell Differentiation, Cell Maturation, Cell Proliferation, Conjugation, Dna Amplification, Dna Methylation, Dna Modification, Gene Conversion, Gene Silencing, Glycosylation, Heteroduplex Formation, Histone Acetylation, Localization, Methylation, Mrna Processing, Regulation Of Gene Expression, Rna Processing, Sensitization, Translation
Quite a number of genes have been found to play important roles in Hemoglobin F Disease, such as ATP6V0A2, BCL11A, CAT, DAND5, GATA1, GPHA2, HBA1, HBB, HBG1, HBG2, KLF1, POU2F1, PSG1, SGCA, SP1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.