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Disease Info Card
Glycogen Storage Disease Type Vii
Information about Glycogen Storage Disease Type Vii: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Glycogen Storage Disease Type Vii
Most recent studies have shown that Glycogen Storage Disease Type Vii shares some biological mechanisms with anemia, anemia-hemolytic, diabetes-mellitus, glycogen-storage-disease, glycogen-storage-disease-type-ii, glycogen-storage-disease-type-iii, glycogen-storage-disease-type-iv, glycogen-storage-disease-type-v, glycogen-storage-disease-type-viii, hemolysis-(disorder), hyperuricemia, muscle-cramp, muscle-weakness, myopathy, pain, rhabdomyolysis, storage-disease, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Glycogen Storage Disease Type Vii, and have been seen in publications frequently: Autophagy, Calcium Ion Homeostasis, Excretion, Fatty Acid Oxidation, Glycolysis, Insulin Secretion, Ion Homeostasis, Localization, Oxidative Phosphorylation, Pathogenesis, Reflex, Reverse Transcription, Rna Splicing, Secretion, Translation, Transport
Quite a number of genes have been found to play important roles in Glycogen Storage Disease Type Vii, such as ACACA, AMPD3, BRAP, CALM1, CALM2, CALM3, CHKA, CHKB, GAA, GBE1, GCK, INS, PFKM, PYGM, QPCT, RPL4, SERPINA3. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.