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Disease Info Card
Familial Partial Lipodystrophy
Information about Familial Partial Lipodystrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Familial Partial Lipodystrophy
Most recent studies have shown that Familial Partial Lipodystrophy shares some biological mechanisms with acanthosis-nigricans, acquired-partial-lipodystrophy, cardiomyopathies, cardiomyopathy-dilated, diabetes-mellitus, diabetes-mellitus-non-insulin-dependent, dyslipidemias, dysplasia, dystrophy, familial-generalized-lipodystrophy, hyperlipidemia, insulin-resistance, lipoatrophic-diabetes-mellitus, lipodystrophy, metabolic-syndrome-x, muscular-dystrophies-limb-girdle, muscular-dystrophy, muscular-dystrophy-emery-dreifuss, obesity.
Among the many pathways, these few ones have gauged particular interests from scientists studying Familial Partial Lipodystrophy, and have been seen in publications frequently: Aging, Cardiac Conduction, Cell Cycle, Cell Differentiation, Chromatin Organization, Chromatin Remodeling, Dna Replication, Glucose Homeostasis, Immune Response, Insulin Secretion, Localization, Mitosis, Nuclear Transport, Pathogenesis, Pigmentation, Secretion, Segmentation, Spermatogenesis, Tissue Development, Transport
Quite a number of genes have been found to play important roles in Familial Partial Lipodystrophy, such as AGPAT2, AMPD1, BANF1, BSCL2, EMD, ERMAP, INS, LBR, LEP, LMNA, LMNB1, LMNB2, MTSS1, PPARA, PPARG, SREBF1, ZMPSTE24. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.