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Disease Info Card
Familial Generalized Lipodystrophy
Information about Familial Generalized Lipodystrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Familial Generalized Lipodystrophy
Most recent studies have shown that Familial Generalized Lipodystrophy shares some biological mechanisms with acanthosis-nigricans, acquired-partial-lipodystrophy, complications-of-diabetes-mellitus, diabetes-mellitus, diabetes-mellitus-non-insulin-dependent, dyslipidemias, fatty-liver, hepatomegaly, hyperinsulinism, hyperlipidemia, hypertrophy, insulin-resistance, lipoatrophic-diabetes-mellitus, lipodystrophy, myeloid-leukemia-chronic, steatohepatitis, steatosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Familial Generalized Lipodystrophy, and have been seen in publications frequently: Adipose Tissue Development, Aging, Cell Death, Energy Homeostasis, Excretion, Glucose Homeostasis, Glycosylation, Hexose Transport, Insulin Secretion, Lipid Homeostasis, Lipid Oxidation, Lipid Storage, Lipoprotein Transport, Localization, Muscle Hypertrophy, Oxidative Phosphorylation, Pathogenesis, Secretion, Tissue Development, Transport
Quite a number of genes have been found to play important roles in Familial Generalized Lipodystrophy, such as ADIPOQ, AGPAT2, BSCL2, CAV1, ERMAP, FOXC2, GGH, GH1, IGF1, INS, INSR, LEP, LMNA, POMC, PPARG, ZMPSTE24. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.