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Disease Info Card
Familial Hypocalciuric Hypercalcaemia
Information about Familial Hypocalciuric Hypercalcaemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Familial Hypocalciuric Hypercalcaemia
Most recent studies have shown that Familial Hypocalciuric Hypercalcaemia shares some biological mechanisms with adenoma, calcium-metabolism-disorders, endocrine-gland-neoplasms, familial-benign-hypercalcemia, hypercalcemia, hypercalciuria, hyperparathyroidism-primary, hyperparathyroidism-secondary, hyperplasia, hypocalcemia, hypoparathyroidism, malignant-neoplasms, multiple-endocrine-neoplasia, multiple-endocrine-neoplasia-type-1, neoplasms, pancreatitis, parathyroid-hyperplasia, parathyroid-neoplasms.
Among the many pathways, these few ones have gauged particular interests from scientists studying Familial Hypocalciuric Hypercalcaemia, and have been seen in publications frequently: Bone Resorption, Cell Growth, Excretion, Glomerular Filtration, Glycosylation, Hormone Secretion, Hypersensitivity, Ion Homeostasis, Localization, Parathyroid Hormone Secretion, Pathogenesis, Protein Secretion, Protein Targeting, Protein Transport, Proteolysis, Secretion, Secretory Pathway, Translation, Transport, Virulence
Quite a number of genes have been found to play important roles in Familial Hypocalciuric Hypercalcaemia, such as CASR, CAT, CDC73, CRAT, CRLF3, GLYAT, IRF9, MEN1, NRGN, PTH, PTRH1, RET, SCTR, SNCG, SRP54, ST13, UCN2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.