Disease Info Card

Dyschondroplasias

Information about Dyschondroplasias: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Dyschondroplasias

Most recent studies have shown that Dyschondroplasias shares some biological mechanisms with angiomatosis, bone-diseases, chondroma, claudication-(finding), congenital-abnormality, dysplasia, enchondromatosis, hemangioma, hypertrophy, lameness-animal, maffucci-syndrome, neoplasms, osteochondritis, osteochondrodysplasias, osteochondrosis, pathologic-neovascularization, poultry-diseases, rickets, swine-diseases, weakness.

Among the many pathways, these few ones have gauged particular interests from scientists studying Dyschondroplasias, and have been seen in publications frequently: Angiogenesis, Bone Development, Bone Mineralization, Cell Death, Cell Differentiation, Cell Proliferation, Chondrocyte Differentiation, Chondrocyte Hypertrophy, Chondrocyte Proliferation, Embryo Development, Endochondral Ossification, Hatching, Limb Development, Localization, Ossification, Pathogenesis, Pigmentation, Regulation Of Growth, Secretion, Transport

Quite a number of genes have been found to play important roles in Dyschondroplasias, such as ACAN, ARSH, DIO3, GRB2, HSP90AA1, IGF1, MMP2, MMP9, PTH, PTHLH, PTRH1, SPP1, TNFSF14, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.