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Disease Info Card
Dental Pulp Necrosis
Information about Dental Pulp Necrosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Dental Pulp Necrosis
Most recent studies have shown that Dental Pulp Necrosis shares some biological mechanisms with abscess, dental-caries, dental-pulp-diseases, dentin-secondary, fracture, gangrene, infective-disorder, inflammation, pain, periapical-diseases, periapical-periodontitis, periodontitis, pulpitis, root-resorption, suppurative-periapical-periodontitis, tooth-avulsion, tooth-fractures, tooth-injuries, tooth-nonvital.
Among the many pathways, these few ones have gauged particular interests from scientists studying Dental Pulp Necrosis, and have been seen in publications frequently: Aging, Apical Constriction, Autolysis, Bone Resorption, Cell Differentiation, Cell Proliferation, Coagulation, Dentinogenesis, Hypersensitivity, Immune Response, Inflammatory Response, Innervation, Localization, Pathogenesis, Regeneration, Root Development, Tissue Regeneration, Transport, Virulence, Wound Healing
Quite a number of genes have been found to play important roles in Dental Pulp Necrosis, such as AAAS, APEX1, BMP7, CALCA, CAT, DBT, FGF2, FUT3, HPS4, IL2, LGI1, PSMD3, RPS3, S100A12, TAC1, TNF, TNFRSF11B, USB1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.