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Disease Info Card
Demyelinating Polyneuropathy
Information about Demyelinating Polyneuropathy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Demyelinating Polyneuropathy
Most recent studies have shown that Demyelinating Polyneuropathy shares some biological mechanisms with autoimmune-diseases, autoimmune-reaction, demyelinating-diseases, demyelination, group-b-streptococcal-infection, guillain-barre-syndrome, inflammation, multiple-sclerosis, nervous-system-disorder, nervousness, peripheral-motor-neuropathy, peripheral-neuropathy, polyneuropathy, polyradiculoneuropathy, polyradiculoneuropathy-chronic-inflammatory-demyelinating, sclerosis, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Demyelinating Polyneuropathy, and have been seen in publications frequently: Cell Adhesion, Cell Death, Cell Differentiation, Cell Proliferation, Complement Activation, Hypersensitivity, Immune Response, Localization, Lymphocyte Proliferation, Mastication, Meiosis, Muscle Atrophy, Muscle Hypertrophy, Myelination, Pathogenesis, Reflex, Regeneration, Secretion, Sensitization, Transport
Quite a number of genes have been found to play important roles in Demyelinating Polyneuropathy, such as CSF2, CST7, CTLA4, DBNL, GDI1, GJB1, IGFALS, LAMC2, MAG, MPZ, MYOM2, PMP22, PREP, PXMP2, RPLP0, SOD1, TNF, TTR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.