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Disease Info Card
Deaf-blind Disorders
Information about Deaf-blind Disorders: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Deaf-blind Disorders
Most recent studies have shown that Deaf-blind Disorders shares some biological mechanisms with absence-of-sensation, blind-vision, cerebral-palsy, cognition-disorders, complete-hearing-loss, depressive-disorder, developmental-disabilities, disorder-of-the-optic-nerve, dystonia-disorders, hearing-loss-bilateral, hearing-problem, impairment-(finding), malignant-neoplasms, optic-atrophy, retinal-degeneration, sensorineural-hearing-loss-(disorder), usher-syndrome, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Deaf-blind Disorders, and have been seen in publications frequently: Aging, Biofilm Formation, Chromatin Remodeling, Cognition, Dna Repair, Eclosion, Localization, Pathogenesis, Phagocytosis, Protein Destabilization, Transport
Quite a number of genes have been found to play important roles in Deaf-blind Disorders, such as BTK, CLRN1, ERCC1, MED12, MYO7A, NDUFB6, OPA1, POLG, POLG2, PRPF6, PRPS1, RPE, TIMM8A, USH1C, USH2A. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.
Pathways Related to Deaf-blind Disorders
This information is being compiled and will come in a future update
| Aging | Biofilm Formation | Chromatin Remodeling |
| Cognition | Dna Repair | Eclosion |
| Localization | Pathogenesis | Phagocytosis |
| Protein Destabilization | Transport |