Disease Info Card

Cutaneous Anaphylaxis (disorder)

Information about Cutaneous Anaphylaxis (disorder): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Cutaneous Anaphylaxis (disorder)

Most recent studies have shown that Cutaneous Anaphylaxis (disorder) shares some biological mechanisms with allergic-rhinitis-(disorder), allergy, anaphylaxis, arthus-reaction, asthma, auricular-swelling, basophilic-leukemia, contact-dermatitis, delayed-hypersensitivity, dermatitis, dermatitis-atopic, edema, eosinophilia, food-allergy, immediate-hypersensitivity, inflammation, leukemia, pertussis, pruritus, rhinorrhea.

Among the many pathways, these few ones have gauged particular interests from scientists studying Cutaneous Anaphylaxis (disorder), and have been seen in publications frequently: Anaphylaxis, Cell Activation, Conjugation, Cytokine Production, Cytokine Secretion, Exocytosis, Fermentation, Humoral Immune Response, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Lymphocyte Proliferation, Mast Cell Activation, Mast Cell Degranulation, Pathogenesis, Phagocytosis, Secretion, Sensitization, Type I Hypersensitivity

Quite a number of genes have been found to play important roles in Cutaneous Anaphylaxis (disorder), such as ALB, C4A, FLVCR1, GRAP2, IFNG, IGHE, IL13, IL4, IL5, IL6, KNG1, MAPK1, NFKB1, SLC17A5, SYK, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.