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Disease Info Card
Complex Regional Pain Syndromes
Information about Complex Regional Pain Syndromes: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Complex Regional Pain Syndromes
Most recent studies have shown that Complex Regional Pain Syndromes shares some biological mechanisms with causalgia, chronic-pain, depressive-disorder, dystonia-disorders, dystrophy, edema, fracture, hyperalgesia, hyperalgesias-mechanical, inflammation, nerve-damage, nervousness, neuralgia, pain, pain-in-limb, pain-intractable, pain-syndrome, peripheral-neuropathy, reflex-sympathetic-dystrophy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Complex Regional Pain Syndromes, and have been seen in publications frequently: Bone Resorption, Cell Differentiation, Complement Activation, Cytokine Production, Glycosylation, Hypersensitivity, Inflammatory Response, Innervation, Localization, Muscle Atrophy, Ossification, Pathogenesis, Platelet Activation, Platelet Aggregation, Reflex, Sensitization, Sensory Processing, Translation, Vasoconstriction, Vasodilation
Quite a number of genes have been found to play important roles in Complex Regional Pain Syndromes, such as C3, CALCA, CD46, CD55, CD59, CRP, CSRP1, GPC3, GRIP1, HLA-DQA1, HNRNPC, IL6, NOD2, S100A12, SGCA, TAC1, TFF2, TNF, TWIST1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.