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- Table of Contents
Information about Chronic Idiopathic Thrombocytopenic Purpura: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Chronic Idiopathic Thrombocytopenic Purpura shares some biological mechanisms with anemia, anemia-hemolytic, autoimmune-diseases, autoimmune-reaction, decreased-immunologic-activity-[pe], helicobacter-infections, helicobacter-pylori-infection, hemorrhage, infective-disorder, intracranial-hemorrhages, leukemia, lupus-erythematosus-systemic, pain, purpura, purpura-thrombocytopenic-idiopathic, purpura-thrombotic-thrombocytopenic, systemic-infection, thrombocytopenic-purpura, thrombocytosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Chronic Idiopathic Thrombocytopenic Purpura, and have been seen in publications frequently: Aging, Cell Activation, Cell Proliferation, Coagulation, Complement Activation, Cytokine Secretion, Hemostasis, Hypersensitivity, Immune Response, Immunoglobulin Production, Interferon-gamma Production, Pathogenesis, Phagocytosis, Platelet Activation, Platelet Aggregation, Programmed Cell Death, Secretion, Sensitization, T Cell Activation, Virulence
Quite a number of genes have been found to play important roles in Chronic Idiopathic Thrombocytopenic Purpura, such as C3, CD4, HP, IL10, IL2, IL4, IL6, ITGA2B, ITGB3, MPL, MS4A1, PGR, RNF130, THPO, TMEM37, TNC, TNF, TPO. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.