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Disease Info Card
Bradykinesia
Information about Bradykinesia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Bradykinesia
Most recent studies have shown that Bradykinesia shares some biological mechanisms with atrophy, cognition-disorders, dementia, depressive-disorder, dyskinetic-syndrome, dystonia-disorders, gait-abnormality, globus-hystericus, hypokinesia, impaired-cognition, movement-disorders, muscle-rigidity, neurodegenerative-disorders, parkinson-disease, progressive-supranuclear-palsy, resting-tremor, secondary-parkinson-disease, supranuclear-palsy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Bradykinesia, and have been seen in publications frequently: Aging, Cell Death, Coagulation, Cognition, Dopamine Uptake, Innervation, Localization, Locomotion, Micturition, Muscle Contraction, Neurogenesis, Neuroprotection, Pathogenesis, Proprioception, Reflex, Regeneration, Secretion, Translation, Transport, Turning Behavior
Quite a number of genes have been found to play important roles in Bradykinesia, such as BPIFA2, COMT, CSF2, DIO2, EEF1A2, GDNF, LRP2, LRRK2, MAPT, MCF2L, MSMB, PSPH, PSPN, REG1A, SIGLEC1, SNCA, STXBP3, TH. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.