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Disease Info Card
Angiokeratoma
Information about Angiokeratoma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Angiokeratoma
Most recent studies have shown that Angiokeratoma shares some biological mechanisms with angiokeratoma-of-fordyce, arthritis, corneal-diseases, dermatologic-disorders, fabry-disease, fucosidase-deficiency-disease, hemangioma, hypertrophy, hypohidrosis, kidney-diseases, kidney-failure, lipoidosis, lysosomal-storage-diseases, melanocytic-nevus, neoplasms, pain, proteinuria-of-undiagnosed-cause, skin-neoplasms, telangiectasis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Angiokeratoma, and have been seen in publications frequently: Aging, Amelogenesis, Excretion, Exocytosis, Gastric Emptying, Glomerular Filtration, Lipid Storage, Localization, Mastication, Methylation, Mrna Splicing, Pathogenesis, Pigmentation, Reverse Transcription, Sensory Perception, Translation, Vasodilation
Quite a number of genes have been found to play important roles in Angiokeratoma, such as AGA, CD34, CTLA4, FOS, GLA, GLB1, HBA1, HLA-DQA1, MANBA, NAGA, NAT8, NOD2, PECAM1, PGR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.