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Disease Info Card
Alpers Syndrome (disorder)
Information about Alpers Syndrome (disorder): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Alpers Syndrome (disorder)
Most recent studies have shown that Alpers Syndrome (disorder) shares some biological mechanisms with ataxia, atrophy, brain-diseases, chronic-progressive-external-ophthalmoplegia, diffuse-cerebral-sclerosis-of-schilder, encephalopathies, epilepsy, external-ophthalmoplegia, intractable-epilepsy, liver-diseases, liver-failure, mitochondrial-diseases, mitochondrial-dna-depletion, myoclonus, myopathy, nerve-degeneration, ophthalmoplegia, status-epilepticus.
Among the many pathways, these few ones have gauged particular interests from scientists studying Alpers Syndrome (disorder), and have been seen in publications frequently: Aging, Cell Death, Cell Proliferation, Dna Replication, Dopamine Uptake, Electron Transport, Electron Transport Chain, Lipid Storage, Liver Regeneration, Mitochondrial Dna Replication, Mitochondrial Translation, Nadh Oxidation, Oxidative Phosphorylation, Pathogenesis, Pyruvate Oxidation, Regeneration, Regulation Of Gene Expression, Translation, Transport, Urea Cycle
Quite a number of genes have been found to play important roles in Alpers Syndrome (disorder), such as CHMP1B, CHP1, COX5A, COX8A, CPOX, CSF2, CYCS, DGUOK, LAMC2, POLG, POLG2, PRNP, PRPF6, SLC25A4, TMED2, TYMP. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.