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Disease Info Card
Adolescent Idiopathic Scoliosis
Information about Adolescent Idiopathic Scoliosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Adolescent Idiopathic Scoliosis
Most recent studies have shown that Adolescent Idiopathic Scoliosis shares some biological mechanisms with abnormal-degeneration, acquired-kyphosis, back-pain, congenital-abnormality, crest-syndrome, curvature-of-spine, hemorrhage, infective-disorder, intraoperative-complications, lordosis, malnutrition, metabolic-bone-disorder, osteopenia, pain, pseudarthrosis, scoliosis-unspecified, spinal-deformity.
Among the many pathways, these few ones have gauged particular interests from scientists studying Adolescent Idiopathic Scoliosis, and have been seen in publications frequently: Aging, Cell Proliferation, Coagulation, Endochondral Ossification, Excretion, Hemostasis, Localization, Locomotion, Menarche, Ossification, Pathogenesis, Platelet Activation, Proprioception, Reflex, Regeneration, Secretion, Segmentation, Swimming, Translation, Transposition
Quite a number of genes have been found to play important roles in Adolescent Idiopathic Scoliosis, such as AR, BEST1, C7, CALM1, CALM2, CALM3, CD6, CERS2, CXCL10, DMD, MTNR1B, RPL3, RPL4, SFPQ, SMS, SS18L1, TP63. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.