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- Table of Contents
, No Gclids
pathway Info Card
Transcription Antitermination
Information about Transcription Antitermination: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Transcription Antitermination
Most recent studies have shown that Transcription Antitermination shares some biological mechanisms with cell-division, cell-growth, cold-acclimation, dna-replication, gene-silencing, nitrate-assimilation, nitrogen-fixation, protein-phosphorylation, regulation-of-gene-expression, rna-folding, rna-polyadenylation, rrna-transcription, sporulation, transcriptional-attenuation, translation, transport, tryptophan-transport, viral-transcription, virion-assembly.
Among the many pathways, these few ones have gauged particular interests from scientists studying Transcription Antitermination, and have been seen in publications frequently: cell-division, cell-growth, cold-acclimation, dna-replication, gene-silencing, nitrate-assimilation, nitrogen-fixation, protein-phosphorylation, regulation-of-gene-expression, rna-folding, rna-polyadenylation, rrna-transcription, sporulation, transcriptional-attenuation, translation, transport, tryptophan-transport, viral-transcription, virion-assembly
Quite a number of genes have been found to play important roles in Transcription Antitermination, such as CHP1, CSH1, DCTN3, DYNC1H1, Dnajc5, EXOSC10, GALK1, GLB1, HOXA10, L1CAM, PHB2, PNLIP, PSMD6, RHO, RPS10, Rhod, TBPL1, TDO2, TYRP1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this pathway. Plesae stay updated.