Disease Info Card

Trigeminal Nerve Injuries

Information about Trigeminal Nerve Injuries: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Trigeminal Nerve Injuries

Most recent studies have shown that Trigeminal Nerve Injuries shares some biological mechanisms with cranial-nerve-diseases, cranial-nerve-injuries, facial-nerve-injuries, facial-pain, fracture, impacted-tooth, intraoperative-complications, lingual-nerve-injuries, malnutrition, mandibular-fractures, neoplasms, nerve-compression-syndrome, nerve-damage, neuralgia, pain, paresthesia, prognathism, sensation-disorders, somatosensory-disorders, trigeminal-neuralgia.

Among the many pathways, these few ones have gauged particular interests from scientists studying Trigeminal Nerve Injuries, and have been seen in publications frequently: Aging, Axon Regeneration, Bone Resorption, Cell Activation, Cell Adhesion, Enucleation, Hypersensitivity, Innervation, Localization, Mastication, Pathogenesis, Reflex, Regeneration, Sensitization, Sensory Perception, Taste Perception, Translation, Transport, Transposition, Wound Healing

Quite a number of genes have been found to play important roles in Trigeminal Nerve Injuries, such as CA9, CAT, CRAT, FUT3, GFAP, GLYAT, GYPA, HPS4, NGF, NPY, NTF4, SLC17A5, SS18L1, TAC1, TH, TRPV1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.