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Disease Info Card
Tay-sachs Disease, Ab Variant
Information about Tay-sachs Disease, Ab Variant: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Tay-sachs Disease, Ab Variant
Most recent studies have shown that Tay-sachs Disease, Ab Variant shares some biological mechanisms with gangliosidoses, gangliosidoses-gm2, hereditary-diseases, infantile-gm>2<-gangliosidosis, lipoidosis, lysosomal-storage-diseases, malignant-neoplasm-of-breast, malnutrition, melanoma, nerve-degeneration, nervous-system-disorder, nervousness, pfaundler-hurler-syndrome, protein-deficiency, sandhoff-disease, storage-disease, tay-sachs-disease, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Tay-sachs Disease, Ab Variant, and have been seen in publications frequently: Aging, Antibody-dependent Cellular Cytotoxicity, Cell Activation, Cell Proliferation, Endocytosis, Excretion, Glycosylation, Interphase, Intracellular Transport, Lipid Storage, Localization, Pathogenesis, Protein Refolding, Startle Response, T Cell Activation, T Cell Proliferation, Translation, Transport
Quite a number of genes have been found to play important roles in Tay-sachs Disease, Ab Variant, such as AKR1C4, BBS9, CTSA, ETFA, GLB1, GM2A, HEXA, HEXB, HHEX, ITGB3, JUN, MS4A1, MTCH1, PGPEP1, PSAP, QPCT, SPANXB1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.