Primary Atelectasis, In Perinatal Period

Overview of Primary Atelectasis, In Perinatal Period

Most recent studies have shown that Primary Atelectasis, In Perinatal Period shares some biological mechanisms with borderline-personality-disorder, bronchopulmonary-dysplasia, chronic-lung-disease, congenital-diaphragmatic-hernia, diaphragmatic-hernia, dysplasia, edema, hemorrhage, hernia, hyaline-membrane-disease, hypoplasia, inflammation, lung-diseases, lung-injury, respiratory-distress, respiratory-distress-syndrome-newborn, respiratory-failure.

Among the many pathways, these few ones have gauged particular interests from scientists studying Primary Atelectasis, In Perinatal Period, and have been seen in publications frequently: Cell Adhesion, Cell Differentiation, Epithelial Cell Differentiation, Exocytosis, Inflammatory Response, Lacrimal Gland Development, Lipid Homeostasis, Localization, Lung Development, Lung Growth, Myelination, Pathogenesis, Phospholipid Homeostasis, Proteolysis, Reverse Transcription, Secretion, Surfactant Homeostasis, Transport, Vasoconstriction, Wound Healing

Quite a number of genes have been found to play important roles in Primary Atelectasis, In Perinatal Period, such as CENPJ, CHDH, FGF7, GLB1, IL10, IL6, MAPK4, MAPK6, MIA, PCNA, SFTPB, SFTPC, SGSM3, TFF2, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Primary Atelectasis, In Perinatal Period Related Genes

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Pathways Related to Primary Atelectasis, In Perinatal Period

This information is being compiled and will come in a future update

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