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Disease Info Card
Nephropathy Toxic
Information about Nephropathy Toxic: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Nephropathy Toxic
Most recent studies have shown that Nephropathy Toxic shares some biological mechanisms with acute-kidney-injury, anemia, carcinoma, infective-disorder, injury-to-kidney, kidney-diseases, kidney-failure, kidney-failure-acute, leukopenia, lung-neoplasms, malignant-neoplasm-of-lung, malignant-neoplasms, nausea, neoplasm-metastasis, neoplasms, proteinuria-of-undiagnosed-cause, renal-insufficiency, vomiting.
Among the many pathways, these few ones have gauged particular interests from scientists studying Nephropathy Toxic, and have been seen in publications frequently: Aging, Angiogenesis, Bone Resorption, Cell Cycle, Cell Death, Cell Proliferation, Coagulation, Conjugation, Diuresis, Excretion, Glomerular Filtration, Hypersensitivity, Immune Response, Localization, Pathogenesis, Regeneration, Secretion, Translation, Transport, Vasoconstriction
Quite a number of genes have been found to play important roles in Nephropathy Toxic, such as ALB, CAT, CTLA4, HLA-DQA1, IL2, MCAT, NAGLU, NBAS, NOD2, PGR, PTGS2, RAPGEF5, SCN7A, SLC17A5, TMEM37, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.