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- Table of Contents
Information about Lipogranuloma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Lipogranuloma shares some biological mechanisms with dermatologic-disorders, eyelid-diseases, granuloma, histiocytoma, histiocytoma-benign-fibrous, histiocytosis, histiocytosis-langerhans-cell, histiocytosis-non-langerhans-cell, inflammation, juvenile-xanthogranuloma, malignant-neoplasms, malignant-paraganglionic-neoplasm, necrobiotic-disorders, necrobiotic-xanthogranuloma, neoplasms, paraproteinemias, skin-neoplasms, xanthoma, xanthomatosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Lipogranuloma, and have been seen in publications frequently: Acute Inflammatory Response, Cell Proliferation, Enucleation, Excretion, Germinal Center Formation, Granuloma Formation, Hemostasis, Immune Response, Inflammatory Response, Lipid Storage, Localization, Mitosis, Ossification, Pathogenesis, Phagocytosis, Pigmentation, Regeneration, Type I Interferon Production, Wound Healing
Quite a number of genes have been found to play important roles in Lipogranuloma, such as BRCA1, CD1A, CD1B, CD1C, CD4, CD68, CTLA4, F13A1, HLA-DQA1, LYZ, NOD2, PTPRC, S100A1, S100A9, S100B, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.