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Disease Info Card
Infantile Globoid Cell Leukodystrophy
Information about Infantile Globoid Cell Leukodystrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Infantile Globoid Cell Leukodystrophy
Most recent studies have shown that Infantile Globoid Cell Leukodystrophy shares some biological mechanisms with atrophy, cerebral-atrophy, demyelination, encephalopathies, gliosis, globoid-cell-leukodystrophy, hypertrophy, leukodystrophy, lipoidosis, muscle-spasticity, nerve-degeneration, nervous-system-disorder, nervousness, neurodegenerative-disorders, optic-atrophy, peripheral-neuropathy, spinal-cord-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Infantile Globoid Cell Leukodystrophy, and have been seen in publications frequently: Cell Death, Glycosylation, Myelination, Pathogenesis
Quite a number of genes have been found to play important roles in Infantile Globoid Cell Leukodystrophy, such as ACAT1, CRAT, CSF2, FANCA, FXN, GALC, GLB1, GLYAT, LAMC2, MBL2, MBP, MTCH1, PRG2, PSAP, SLC25A5. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.
Pathways Related to Infantile Globoid Cell Leukodystrophy
This information is being compiled and will come in a future update
| Cell Death | Glycosylation | Myelination |
| Pathogenesis |