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Disease Info Card
Hypophosphatemic Rickets
Information about Hypophosphatemic Rickets: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hypophosphatemic Rickets
Most recent studies have shown that Hypophosphatemic Rickets shares some biological mechanisms with autosomal-dominant-hypophosphatemic-rickets, bone-diseases, congenital-abnormality, dwarfism, familial-hypophosphatemia, familial-hypophosphatemic-bone-disease, fanconi-syndrome, hypercalcemia, hypercalciuria, hypophosphatemia, kidney-diseases, neoplasms, nephrocalcinosis, oncogenic-osteomalacia, osteomalacia, phosphate-diabetes, rickets, vitamin-d-deficiency.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hypophosphatemic Rickets, and have been seen in publications frequently: Bone Development, Bone Mineralization, Bone Resorption, Cell Proliferation, Dentin Mineralization, Dentinogenesis, Excretion, Glomerular Filtration, Glycosylation, Growth Hormone Secretion, Intestinal Absorption, Localization, Ossification, Osteoblast Differentiation, Pathogenesis, Renal Phosphate Excretion, Secretion, Sodium-dependent Phosphate Transport, Translation, Transport
Quite a number of genes have been found to play important roles in Hypophosphatemic Rickets, such as AVPR2, BGLAP, CLCN5, DMP1, DMTF1, FGF23, GGH, GH1, GSTP1, MEPE, PHEX, PTH, PTRH1, RAPGEF5, SERPINA1, SLC34A1, SLC34A3. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.