Hydrops Fetalis, Non-immune

Overview of Hydrops Fetalis, Non-immune

Most recent studies have shown that Hydrops Fetalis, Non-immune shares some biological mechanisms with anemia, cytogenetic-abnormality, disease-due-to-parvoviridae, edema, erythema-infectiosum, fetal-death, fetal-diseases, heart-diseases, heart-failure, hydrops-fetalis, hypoplasia, lymphangioma-cystic, parvovirus-b19-(disease), pleural-effusion-disorder, polyhydramnios, pregnancy-complications, pregnancy-complications-infectious, pulmonary-hypoplasia.

Among the many pathways, these few ones have gauged particular interests from scientists studying Hydrops Fetalis, Non-immune, and have been seen in publications frequently: Bone Maturation, Glycosylation, Immune Response, Lipid Storage, Lung Growth, Lymphangiogenesis, Ossification, Pathogenesis, Protein Import, Transposition, Tropism

Quite a number of genes have been found to play important roles in Hydrops Fetalis, Non-immune, such as AFP, ALB, CCBE1, EVA1C, GATA1, GBA, GLB1, GUSB, HBA1, HUNK, IVNS1ABP, PLOD1, PRL, PSMD4, PTPN11, RHCE, RHD. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Hydrops Fetalis, Non-immune Related Genes

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Pathways Related to Hydrops Fetalis, Non-immune

This information is being compiled and will come in a future update

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