Disease Info Card

Fibromatosis, Aggressive

Information about Fibromatosis, Aggressive: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Fibromatosis, Aggressive

Most recent studies have shown that Fibromatosis, Aggressive shares some biological mechanisms with abdominal-neoplasms, adenomatous-polyposis-coli, bone-neoplasms, carcinoma, fibroma, fibromatosis, fibromatosis-abdominal, fibrosarcoma, gardner-syndrome, malignant-neoplasms, malignant-paraganglionic-neoplasm, neoplasm-metastasis, neoplasm-recurrence-local, neoplasms, pain, polyposis, recurrence-(disease-attribute), sarcoma, soft-tissue-neoplasms, thoracic-neoplasms.

Among the many pathways, these few ones have gauged particular interests from scientists studying Fibromatosis, Aggressive, and have been seen in publications frequently: Angiogenesis, Cell Adhesion, Cell Cycle, Cell Growth, Cell Migration, Cell Motility, Cell Proliferation, Dehiscence, Fibroblast Proliferation, Invasive Growth, Localization, Methylation, Mismatch Repair, Mitosis, Oncogenesis, Ossification, Pathogenesis, Transposition, Wound Healing

Quite a number of genes have been found to play important roles in Fibromatosis, Aggressive, such as APC, CD34, CEL, CTNNB1, DCT, DES, DST, FAP, GLMN, KIT, PGR, PROC, TNF, TP53, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.