Disease Info Card

Chronic Eosinophilic Inflammation

Information about Chronic Eosinophilic Inflammation: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Chronic Eosinophilic Inflammation

Most recent studies have shown that Chronic Eosinophilic Inflammation shares some biological mechanisms with abdominal-pain, allergy, asthma, edema, eosinophilia, eosinophilic-esophagitis, eosinophilic-gastroenteritis, esophagitis, fibrosis, gastroenteritis, granuloma, hypereosinophilic-syndrome, hyperplasia, inflammation, neoplasms, pain, pneumonia, polyps, rhinorrhea, vasculitis.

Among the many pathways, these few ones have gauged particular interests from scientists studying Chronic Eosinophilic Inflammation, and have been seen in publications frequently: Anaphylaxis, Cell Activation, Cell Adhesion, Cell Proliferation, Chemotaxis, Cytokine Production, Eosinophil Activation, Eosinophil Degranulation, Granuloma Formation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Mast Cell Degranulation, Muscle Hypertrophy, Parturition, Pathogenesis, Peristalsis, Secretion, Sensitization

Quite a number of genes have been found to play important roles in Chronic Eosinophilic Inflammation, such as AHR, CCL11, CCL2, CCL5, CD4, CD8A, CSF2, GNAI1, IFNG, IGHE, IL10, IL13, IL2, IL4, IL5, IL6, RNASE3, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.