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- Table of Contents
Information about Brittle Hair: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Brittle Hair shares some biological mechanisms with cockayne-syndrome, dermatologic-disorders, developmental-delay-(disorder), dwarfism, dysplasia, ectodermal-dysplasia, growth-disorders, growth-retardation, hair-diseases, hereditary-diseases, ichthyoses, malignant-neoplasm-of-skin, malignant-neoplasms, photosensitivity-disorders, trichothiodystrophy-syndromes, xeroderma, xeroderma-pigmentosum, xeroderma-pigmentosum-group-d.
Among the many pathways, these few ones have gauged particular interests from scientists studying Brittle Hair, and have been seen in publications frequently: Aging, Anagen, Cell Cycle, Cell Cycle Arrest, Cell Killing, Cell Proliferation, Cellular Response To Uv, Dna Excision, Dna Repair, Excretion, Hypersensitivity, Myelination, Nucleotide-excision Repair, Pathogenesis, Pigmentation, Regeneration, Response To Uv, Tissue Development, Transport, Uv Protection
Quite a number of genes have been found to play important roles in Brittle Hair, such as ADAM17, ASL, CBS, CLDN3, CS, ERCC1, ERCC2, ERCC3, ERCC4, GTF2H1, GTF2H2, GTF2H3, GTF2H4, GTF2H5, IFNA1, MPLKIP, NR1H2, STOM, TBPL1, TYRP1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.