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Disease Info Card
Ameloblastic Fibroma
Information about Ameloblastic Fibroma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Ameloblastic Fibroma
Most recent studies have shown that Ameloblastic Fibroma shares some biological mechanisms with adenomatoid-odontogenic-tumor, ameloblastic-fibro-odontoma, ameloblastic-fibrosarcoma, ameloblastoma, calcifying-odontogenic-cyst, dentinogenic-ghost-cell-tumor, fibroma, fibrosarcoma, hamartoma, jaw-neoplasms, malignant-paraganglionic-neoplasm, mandibular-neoplasms, maxillary-neoplasms, neoplasm-recurrence-local, neoplasms, odontogenic-cysts, odontogenic-fibroma, odontogenic-tumors, odontoma.
Among the many pathways, these few ones have gauged particular interests from scientists studying Ameloblastic Fibroma, and have been seen in publications frequently: Bone Resorption, Cell Adhesion, Cell Proliferation, Dentinogenesis, Enucleation, Invasive Growth, Keratinization, Localization, Odontogenesis, Ossification, Pathogenesis, Phagocytosis, Pigmentation, Tooth Eruption
Quite a number of genes have been found to play important roles in Ameloblastic Fibroma, such as AMBN, AMTN, CAT, CRAT, DES, ENO1, ENO2, FN1, GFAP, GLYAT, KRT14, KRT7, ODAM, PCNA, PSMD4, TNC, TNFRSF11A, TP53, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.