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Disease Info Card
Xerophthalmia
Information about Xerophthalmia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Xerophthalmia
Most recent studies have shown that Xerophthalmia shares some biological mechanisms with autoimmune-diseases, autoimmune-reaction, blind-vision, conjunctival-diseases, corneal-diseases, corneal-ulcer, disorder-of-eye, dry-eye-syndromes, keratoconjunctivitis, malnutrition, night-blindness, nutrition-disorders, primary-sjã¶grens-syndrome, salivary-gland-diseases, sicca-syndrome, sjogrens-syndrome, trachoma, vitamin-a-deficiency, xerostomia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Xerophthalmia, and have been seen in publications frequently: Aging, Cell Differentiation, Cell Proliferation, Enucleation, Excretion, Hypersensitivity, Immune Response, Innervation, Keratinization, Lactation, Pathogenesis, Pigmentation, Protein Secretion, Reflex, Response To Vitamin, Response To Vitamin A, Secretion, Tear Secretion, Transport, Transposition
Quite a number of genes have been found to play important roles in Xerophthalmia, such as AAAS, ALB, ATN1, CALR, CIC, CRP, IL4, IL6, LYZ, POMC, RBP4, RENBP, SSB, SUGP1, TNF, TRIM21. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.