This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Trichostrongylosis
Information about Trichostrongylosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Trichostrongylosis
Most recent studies have shown that Trichostrongylosis shares some biological mechanisms with atrophy, bird-diseases, cattle-diseases, diarrhea, eosinophilia, gastroenteritis, gastrointestinal-diseases, goat-diseases, helminthiasis, helminthiasis-animal, hookworm-infections, infective-disorder, intestinal-diseases-parasitic, nematode-infections, oesophagostomiasis, sheep-diseases, trematode-infections, trichostrongyloidiasis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Trichostrongylosis, and have been seen in publications frequently: Cell Migration, Drug Resistance, Eosinophil Differentiation, Excretion, Fermentation, Hatching, Hypersensitivity, Immune Response, Inflammatory Response, Lactation, Larval Development, Lymphocyte Proliferation, Mucosal Immune Response, Parasitism, Parturition, Pathogenesis, Response To Nematode, Secretion, Transport, Tricarboxylic Acid Cycle
Quite a number of genes have been found to play important roles in Trichostrongylosis, such as ACHE, ALB, CERS2, DMPK, GNAI1, HM13, IFNG, IGHE, IL10, IL5, LIPF, OSTM1, PPP1R3B, RASIP1, RPL3, RPL4, SMCP, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.