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Disease Info Card
Pancreatic Acinar Atrophy
Information about Pancreatic Acinar Atrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Pancreatic Acinar Atrophy
Most recent studies have shown that Pancreatic Acinar Atrophy shares some biological mechanisms with aneurysm, aneurysm-of-popliteal-artery, atrophy, carcinoma, depressive-disorder, diarrhea, edema, escherichia-coli-infections, hemorrhage, hypertensive-disease, infective-disorder, inflammation, ischemia, malignant-neoplasms, neoplasm-metastasis, neoplasms, pulmonary-artery-aneurysm, thrombosis, tissue-adhesions.
Among the many pathways, these few ones have gauged particular interests from scientists studying Pancreatic Acinar Atrophy, and have been seen in publications frequently: Cell Adhesion, Cell Cycle, Cell Growth, Cell Proliferation, Coagulation, Conjugation, Dna Replication, Excretion, Fermentation, Fibrinolysis, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Pathogenesis, Platelet Aggregation, Secretion, Transport, Viral Replication, Virulence
Quite a number of genes have been found to play important roles in Pancreatic Acinar Atrophy, such as AFM, ALB, AMY2A, BLOC1S6, CDSN, CS, FN1, INS, MUC1, PAH, PLAT, PLAU, PLG, POLG, SERPINE1, TEAD1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.