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Disease Info Card
Osteolysis
Information about Osteolysis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Osteolysis
Most recent studies have shown that Osteolysis shares some biological mechanisms with arthritis, bone-diseases, bone-neoplasms, degenerative-polyarthritis, dislocations, fracture, hypercalcemia, malignant-neoplasm-of-breast, malignant-neoplasms, malignant-paraganglionic-neoplasm, mammary-neoplasms, metastatic-malignant-neoplasm-to-the-bone, multiple-myeloma, neoplasm-metastasis, neoplasms, osteolysis-essential, osteopenia, osteoporosis, pain, periprosthetic-osteolysis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Osteolysis, and have been seen in publications frequently: Aging, Angiogenesis, Bone Remodeling, Bone Resorption, Cell Growth, Cell Proliferation, Cytokine Production, Excretion, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Macrophage Activation, Ossification, Osteoblast Differentiation, Osteoclast Differentiation, Pathogenesis, Phagocytosis, Regeneration, Secretion
Quite a number of genes have been found to play important roles in Osteolysis, such as ACP5, BGLAP, CALCA, CD40LG, IL1B, IL6, NFKB1, PTH, PTHLH, SCYL1, TDRD7, TNF, TNFRSF11A, TNFRSF11B, TNFSF11, TRAF2, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.