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Disease Info Card
Ophthalmopareses
Information about Ophthalmopareses: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Ophthalmopareses
Most recent studies have shown that Ophthalmopareses shares some biological mechanisms with ataxia, atrophy, blepharoptosis, chronic-progressive-external-ophthalmoplegia, diplopia, dysarthria, external-ophthalmoplegia, group-b-streptococcal-infection, guillain-barre-syndrome, miller-fisher-syndrome, mitochondrial-diseases, myopathy, nerve-paralysis, nystagmus, ophthalmoplegia, pain, peripheral-neuropathy, sclerosis, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Ophthalmopareses, and have been seen in publications frequently: Blood Circulation, Cardiac Conduction, Cell Growth, Coagulation, Dna Replication, Localization, Locomotion, Mitochondrial Dna Replication, Muscle Atrophy, Muscle Hypertrophy, Oxidative Phosphorylation, Pathogenesis, Pigmentation, Reflex, Regeneration, Regulation Of Gene Expression, Synaptic Transmission, System Process, Translation, Transport
Quite a number of genes have been found to play important roles in Ophthalmopareses, such as ACHE, AFG3L2, ATXN1, CHMP1B, COX5A, COX8A, CPOX, CSF2, CYCS, DNM2, IGFALS, LAMC2, LCN1, MTM1, POLG, SOD1, TMPO, TYMP. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.