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Disease Info Card
Inclusion Conjunctivitis
Information about Inclusion Conjunctivitis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Inclusion Conjunctivitis
Most recent studies have shown that Inclusion Conjunctivitis shares some biological mechanisms with bacterial-conjunctivitis, chlamydia-infections, conjunctival-diseases, conjunctivitis, eye-infections, female-genital-diseases, gonorrhea, infective-disorder, keratoconjunctivitis, lymphogranuloma-venereum, ophthalmia-neonatorum, pneumonia, pregnancy-complications-infectious, psittacosis, sexually-transmitted-diseases, trachoma, urethritis, uterine-cervicitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Inclusion Conjunctivitis, and have been seen in publications frequently: Acute Inflammatory Response, Antigenic Variation, Cell Division, Chemokine Production, Dna Amplification, Endocytosis, Estrus, Humoral Immune Response, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Mating, Pathogenesis, Phagocytosis, Secretion, Swimming, Translation, Transport, Virulence
Quite a number of genes have been found to play important roles in Inclusion Conjunctivitis, such as BAX, CCL5, CD8A, CHP1, CRAT, HSPD1, IL2, MARVELD2, MIF, PPFIBP1, RB1, S100A8, S100A9, TLR2, TNF, ZACN. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.