Disease Info Card

Aggressive Systemic Mastocytosis

Information about Aggressive Systemic Mastocytosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Aggressive Systemic Mastocytosis

Most recent studies have shown that Aggressive Systemic Mastocytosis shares some biological mechanisms with asthma, bronchial-hyperreactivity, chronic-obstructive-airway-disease, dermatitis, dermatitis-atopic, fibrosis, hyperplasia, hypertrophy, infective-disorder, inflammation, leukemia, lung-diseases, lung-diseases-obstructive, malignant-neoplasms, mastocytosis, mastocytosis-systemic, neoplasms, niemann-pick-diseases, observation-of-neuromuscular-block, tissue-adhesions.

Among the many pathways, these few ones have gauged particular interests from scientists studying Aggressive Systemic Mastocytosis, and have been seen in publications frequently: Aging, Angiogenesis, Cell Activation, Cell Adhesion, Cell Cycle, Cell Death, Cell Growth, Cell Migration, Cell Proliferation, Inflammatory Response, Localization, Muscle Cell Proliferation, Muscle Contraction, Pathogenesis, Secretion, Segmentation, Sensitization, Smooth Muscle Cell Proliferation, Smooth Muscle Contraction, Transport

Quite a number of genes have been found to play important roles in Aggressive Systemic Mastocytosis, such as AHR, CCL11, EGF, EPHB2, IL13, IL4, IL5, IL6, KIT, KNG1, MAPK1, MAPK3, PIK3CG, PSMC6, SMPD1, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.