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Disease Info Card
Sjogren-larsson Syndrome
Information about Sjogren-larsson Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Sjogren-larsson Syndrome
Most recent studies have shown that Sjogren-larsson Syndrome shares some biological mechanisms with congenital-ichthyosis, congenital-nonbullous-ichthyosiform-erythroderma, diplegia, dystrophy, epilepsy, exfoliative-dermatitis, hereditary-diseases, hyperkeratosis, ichthyoses, ichthyosis-x-linked, littles-disease, muscle-spasticity, neurocutaneous-syndromes, quadriplegia, refsum-disease, sjogrens-syndrome, streptococcal-lymphadenitis-of-swine.
Among the many pathways, these few ones have gauged particular interests from scientists studying Sjogren-larsson Syndrome, and have been seen in publications frequently: Autolysis, Blood Circulation, Cell Adhesion, Cell Cycle, Cell Growth, Cell Proliferation, Cornification, Hypersensitivity, Inflammatory Response, Keratinization, Light Absorption, Lipid Storage, Localization, Membrane Organization, Myelination, Pathogenesis, Protein Folding, Protein Import, Regulation Of Cell Growth, Translation
Quite a number of genes have been found to play important roles in Sjogren-larsson Syndrome, such as ADH5, AKR1A1, ALDH3A1, ALDH3A2, CD19, CYP4F2, FAH, FANCA, HSPB1, IKBKG, LTA, LTB, PLXNA2, PPARA, QPCT. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.