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Disease Info Card
Scimitar Syndrome
Information about Scimitar Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Scimitar Syndrome
Most recent studies have shown that Scimitar Syndrome shares some biological mechanisms with anomalous-pulmonary-venous-connection, atresia, atrial-septal-defects, congenital-abnormality, congenital-heart-defects, congenital-heart-disease, heart-diseases, heart-failure, heart-septal-defects, hypertensive-disease, hypoplasia, partial-anomalous-pulmonary-venous-return, patent-ductus-arteriosus, pulmonary-hypertension, stenosis, tetralogy-of-fallot, transposition-of-great-vessels, venous-occlusion, ventricular-septal-defects.
Among the many pathways, these few ones have gauged particular interests from scientists studying Scimitar Syndrome, and have been seen in publications frequently: Aging, Cell Differentiation, Cell Proliferation, Coagulation, Dehiscence, Endothelial Cell Proliferation, Hemostasis, Localization, Methylation, Myelination, Ossification, Pathogenesis, Reflex, Segmentation, Tissue Migration, Transport, Transposition, Tube Fusion, Vasculogenesis, Vasoconstriction
Quite a number of genes have been found to play important roles in Scimitar Syndrome, such as AMY2A, ARSD, ASAP2, BCHE, BLOC1S6, CELA3B, CPB1, ENOPH1, EPB42, GDF1, GLYAT, MRPS30, PAPOLA, PDAP1, PRH1, REG3A, TUSC2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.