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Disease Info Card
Infectious Bovine Keratoconjunctivitis
Information about Infectious Bovine Keratoconjunctivitis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Infectious Bovine Keratoconjunctivitis
Most recent studies have shown that Infectious Bovine Keratoconjunctivitis shares some biological mechanisms with bacterial-infections, cattle-diseases, cell-invasion, corneal-diseases, dysequilibrium-syndrome, fusariosis, infective-disorder, keratoconjunctivitis, keratoconjunctivitis-infectious, mucocele-of-mouth, mycoses, plant-diseases, scab, steroid-sulfatase-deficiency-disease, tetraploidy, tissue-adhesions, ulcer, visual-halo.
Among the many pathways, these few ones have gauged particular interests from scientists studying Infectious Bovine Keratoconjunctivitis, and have been seen in publications frequently: Asexual Sporulation, Cell Death, Colony Morphology, Defense Response, Disease Resistance, Gene Silencing, Germination, Hyphal Growth, Localization, Mating, Pathogenesis, Pigmentation, Programmed Cell Death, Secretion, Sexual Reproduction, Sporulation, Systemic Acquired Resistance, Translation, Transport, Virulence
Quite a number of genes have been found to play important roles in Infectious Bovine Keratoconjunctivitis, such as ATP6V0A1, ATP6V0A2, CACFD1, F2, GPHA2, PGR, PLXNA1, PRDX6, PRTN3, RPL10, RPS4X, SGCA, STS, SYF2, TMEM37, TTF2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.