Sideroflexin-4 Antibodies

AND Sfxn4 ELISA kits, Sideroflexin-4 Proteins

Many biological assays use antibodies to detect Sideroflexin-4, such as Western Blot (WB), ELISA, Flow Cytometry, Immunocytochemistry (IHC). These antibodies can be polyclonal or monoclonal, and react with Sideroflexin-4 in Human, Mouse, Rat, and many other animal samples. Boster Bio use mainly mouse and rabbit to develop Sideroflexin-4 antibodies...

We validate the specificity of these antibodies to Sideroflexin-4 by testing them on tissues known to express Sfxn4 positively and negatively. Browse below to find the Sfxn4 antibody that suites your experiment. We have 2 of these antibodies and many publications and validation images.

If you cannot find antibodies that fit your needs, contact us for making custom antibodies. We have a full suite of custom antibody services covering from research to diagnostic and therapeutic applications.

Previous: Septin9 Next: Cadm3

Sfxn4 Infographic by Boster Bio

All Sfxn4 Products

Product

Figures

Specificity

Applications

Price

Anti-Sideroflexin-4 SFXN4 Antibody

7 Q&As

Human

$360
Cat # A12029
100 µl

SFXN4 (NM_213649) Human Over-expression Lysate

Human

$628
Cat # LS119559
100 µg

Sfxn4 Overview

Facts about Sideroflexin-4.

Sideroflexin-4 (Sfxn4)

Potential iron transporter. .

Gene Information

Mouse
Gene Name:	Sfxn4
Uniprot:	Q925N1
Entrez:	94281

Family

Belongs to:

sideroflexin family

Alternative Names

BCRM1; Breast cancer resistance marker 1; sideroflexin 4; sideroflexin-4

Molecular Weight

Mass (kDA):
35.58 kDA

Genomic Context


Mouse

Location:	19\|19 D3
Sequence:	19;

Tissue Specificity

Largely restricted to kidney, brain and heart.

Diseases

Malignant Neoplasms
Malignant Neoplasm Of Breast
Aniridia
Anemia
Nervousness
Liver Carcinoma
Renal Cell Carcinoma
Pemphigoid, Benign Mucous Membrane
Liver Cirrhosis

Bullous Pemphigoid
Sideroblastic Anemia
Carcinoma

Pathways

Transport

PTMs

Acetylation

For details, visit:
bosterbio.com/bosterbio-gene-info-cards/Sfxn4

References

PMID: 11274051 by Fleming M.D., et al. A mutation in a mitochondrial transmembrane protein is responsible for the pleiotropic hematological and skeletal phenotype of flexed- tail (f/f) mice.