Sideroflexin-2 Antibodies

AND Sfxn2 ELISA kits, Sideroflexin-2 Proteins

Many biological assays use antibodies to detect Sideroflexin-2, such as Western Blot (WB), ELISA, Flow Cytometry, Immunocytochemistry (IHC). These antibodies can be polyclonal or monoclonal, and react with Sideroflexin-2 in Human, Mouse, Rat, and many other animal samples. Boster Bio use mainly mouse and rabbit to develop Sideroflexin-2 antibodies...

We validate the specificity of these antibodies to Sideroflexin-2 by testing them on tissues known to express Sfxn2 positively and negatively. Browse below to find the Sfxn2 antibody that suites your experiment. We have 2 of these antibodies and many publications and validation images.

If you cannot find antibodies that fit your needs, contact us for making custom antibodies. We have a full suite of custom antibody services covering from research to diagnostic and therapeutic applications.

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Sfxn2 Infographic by Boster Bio

All Sfxn2 Products

Product

Figures

Specificity

Applications

Price

SFXN2 (NM_178858) Human Recombinant Protein

$1249
Cat # PROTQ96NB2
20 µg

SFXN2 (NM_178858) Human Over-expression Lysate

Human

$628
Cat # LS118980
100 µg

Sfxn2 Overview

Facts about Sideroflexin-2.

Sideroflexin-2 (Sfxn2)

Potential iron transporter. .

Gene Information

Mouse
Gene Name:	Sfxn2
Uniprot:	Q925N2
Entrez:	94279

Family

Belongs to:

sideroflexin family

Alternative Names

sideroflexin 2; sideroflexin-2

Molecular Weight

Mass (kDA):
36.141 kDA

Genomic Context


Mouse

Location:	19\|19 C3
Sequence:	19;

Tissue Specificity

Expressed in brain, heart, kidney, spleen, thymus, liver, stomach and skin.

Diseases

Anemia
Insulinoma

Pathways

Regeneration
Sperm Motility
Insulin Secretion
Fertilization
Secretion

PTMs

Acetylation

For details, visit:
bosterbio.com/bosterbio-gene-info-cards/Sfxn2

References

PMID: 11274051 by Fleming M.D., et al. A mutation in a mitochondrial transmembrane protein is responsible for the pleiotropic hematological and skeletal phenotype of flexed- tail (f/f) mice.