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Disease Info Card
Fazio-londe Syndrome
Information about Fazio-londe Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Fazio-londe Syndrome
Most recent studies have shown that Fazio-londe Syndrome shares some biological mechanisms with amyotrophic-lateral-sclerosis, atrophy, bulbar-palsy-progressive, bulbar-palsy-progressive-with-sensorineural-deafness, complete-hearing-loss, deglutition-disorders, facial-paresis, motor-neuron-disease, nervous-system-disorder, neuromuscular-diseases, paresis, primary-lateral-sclerosis, riboflavin-deficiency, sclerosis, sensorineural-hearing-loss-(disorder), spinal-muscular-atrophy, stridor, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Fazio-londe Syndrome, and have been seen in publications frequently:
Quite a number of genes have been found to play important roles in Fazio-londe Syndrome, such as ETFA, ETFB, ETFDH, MADD, SLC52A1, SLC52A2, SLC52A3. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.