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- Table of Contents
Facts about Zinc finger protein AEBP2.
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Mouse | |
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Gene Name: | Aebp2 |
Uniprot: | Q9Z248 |
Entrez: | 11569 |
Belongs to: |
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AEBP2/jing C2H2-type zinc-finger family |
Adipocyte enhancer-binding protein 2; AE binding protein 2; AE(adipocyte enhancer)-binding protein 2; AE-binding protein 2; MGC17922; zinc finger protein AEBP2
Mass (kDA):
52.963 kDA
Mouse | |
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Location: | 6|6 G2 |
Sequence: | 6; |
Expressed in brain, brown adipose tissue, white adipose tissue, heart, kidney, lung, skeletal muscle, small intestine and spleen. Expressed at low levels in liver.
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The AEBP2 RNA molecule plays a role in the cellular immune response to RABV. Six RABV-infected N2a cell lines were infected at a MOI 0.01, with six lncRNAs strongly upregulated. XLOC_007537 is 1564 nt in length and was predicted to be transcribed at an intergenic locus on chromosome 11. It was highly induced and inhibited RABV infections in N2a cells.
The AEBP2 gene was found in the nuclei of RABV virus and is associated with a high level of transcription. The EDALEZH2 enzyme transcribes the protein and regulates its expression. This gene is critical for the immune reaction to RABV infection. Drug discovery is a priority. This gene can be used to identify RABVN proteins in real-time. It can also be used to monitor and treat viral infections.
RABVEDAL virus can cause a number inflammatory or innate immunity-related genetic mutations in human brains. CE infection, however, is not fatal. The CE(NiN), strain contains the Ni-derived N gene. This may be due to the difference in pathogenicity among these two strains. Its role is not yet known, but it will aid scientists and physicians in making better decisions about vaccines.
The infection process is facilitated by the AEBP2 protein gene. RABV N protein is produced from viral genomic RNA. When intranasal, EDAL prevents RABV infection. In mice, it also inhibits RABV-induced intranasal infection. Similar studies showed that dsRNA as well as interferons can induce EDAL.
This gene is involved by the immune system not responding to the antiviral threat. It is involved in the regulation and expression of genes related innate immunity and the inflammatory response. Ni N avoids induced antiviral effects in adult mice. This gene regulates RIG-I expression and helps prevent the development AIDS.
Viral replication is also influenced by the AEBP2 Gene. It serves as a template for RNA synthesis and replication. Protein N binds to a protein called protein P during viral replication. This prevents its phosphorylation. Protein N also protects the genome from nucleases. The gene encodes many protein products, including M proteins and N proteins.
The RABV infection marker is the AEBP2 genetic gene. Using this gene in patients is an important step in the diagnosis of the disease and monitoring progress. It has the potential of improving patient care by detecting or preventing the virus from replication. It is also a valuable diagnostic tool for evaluating the severity of RABV infection.
The RABV virus can be considered a neurotropic viral. To reach the brain, the virus uses stealth. While this strategy works, it is not widely used. This is because there are many untapped opportunities for the AEBP2 RABV marker. Further research and development will be required to develop an effective treatment and vaccine against RABV.
PMID: 10329662 by He G.-P., et al. Cloning and characterization of a novel zinc finger transcriptional repressor. A direct role of the zinc finger motif in repression.
PMID: 19026780 by Shen X., et al. EZH1 mediates methylation on histone H3 lysine 27 and complements EZH2 in maintaining stem cell identity and executing pluripotency.