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Disease Info Card
Spinocerebellar Ataxia Type 7
Information about Spinocerebellar Ataxia Type 7: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Spinocerebellar Ataxia Type 7
Most recent studies have shown that Spinocerebellar Ataxia Type 7 shares some biological mechanisms with abnormal-degeneration, age-related-macular-degeneration, ataxia, ataxia-spinocerebellar, atrophy, blind-vision, cerebellar-ataxia, cerebellar-diseases, dystrophy, huntington-disease, nerve-degeneration, nervous-system-disorder, neurodegenerative-disorders, retinal-degeneration, retinal-diseases, retinitis-pigmentosa, spinocerebellar-degeneration, trinucleotide-repeat-expansion, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Spinocerebellar Ataxia Type 7, and have been seen in publications frequently: Autophagy, Cell Death, Cell Division, Cell Migration, Chromatin Remodeling, Dna Conformation Change, Gene Silencing, Glycosylation, Histone Acetylation, Localization, Macroautophagy, Methylation, Neuron Death, Nuclear Export, Pathogenesis, Proteolysis, Reflex, Rna Interference, Translation, Transport
Quite a number of genes have been found to play important roles in Spinocerebellar Ataxia Type 7, such as ATXN1, ATXN3, ATXN7, CACNA1A, CASP3, CREBBP, CRX, ERG, GLB1, HSPA4, HTT, KAT2A, KCNH2, PDYN, PML, SORBS1, SUPT3H, TBP. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.