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Disease Info Card
Mycobacterium Avium-intracellulare Infection
Information about Mycobacterium Avium-intracellulare Infection: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Mycobacterium Avium-intracellulare Infection
Most recent studies have shown that Mycobacterium Avium-intracellulare Infection shares some biological mechanisms with acquired-immunodeficiency-syndrome, aids-related-opportunistic-infections, bacteremia, bronchiectasis, cytomegalovirus-infections, granuloma, hiv-infections, immunologic-deficiency-syndromes, infective-disorder, kaposi-sarcoma, lung-diseases, lymphadenitis, mycobacterium-infections, mycobacterium-infections-nontuberculous, opportunistic-infections, pneumonia, pneumonia-pneumocystis-carinii, tuberculosis, tuberculosis-pulmonary.
Among the many pathways, these few ones have gauged particular interests from scientists studying Mycobacterium Avium-intracellulare Infection, and have been seen in publications frequently: Cell Proliferation, Colony Morphology, Cytokine Production, Drug Resistance, Excretion, Granuloma Formation, Hypersensitivity, Immune Response, Inflammatory Response, Interferon-gamma Production, Localization, Macrophage Activation, Pathogenesis, Phagocytosis, Response To Drug, Secretion, Swimming, Transport, Viral Replication, Virulence
Quite a number of genes have been found to play important roles in Mycobacterium Avium-intracellulare Infection, such as CALM1, CALM2, CALM3, CAMKMT, CD4, CSF2, FPR2, GSTZ1, HSPD1, IFNG, IL10, IL2, IL6, KRIT1, NTM, SLC11A1, TNF, TRIM27. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.