SLC38A8 Antibodies

AND SLC38A8 ELISA kits, SLC38A8 Proteins

Many biological assays use antibodies to detect SLC38A8, such as Western Blot (WB), ELISA, Flow Cytometry, Immunocytochemistry (IHC). These antibodies can be polyclonal or monoclonal, and react with SLC38A8 in Human, Mouse, Rat, and many other animal samples. Boster Bio use mainly mouse and rabbit to develop SLC38A8 antibodies...

We validate the specificity of these antibodies to SLC38A8 by testing them on tissues known to express SLC38A8 positively and negatively. Browse below to find the SLC38A8 antibody that suites your experiment. We have 1 of these antibodies and many publications and validation images.

If you cannot find antibodies that fit your needs, contact us for making custom antibodies. We have a full suite of custom antibody services covering from research to diagnostic and therapeutic applications.

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SLC38A8 Infographic by Boster Bio

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SLC38A8 (NM_001080442) Human Over-expression Lysate

Human

$960
Cat # LS146167
100 µg

SLC38A8 Overview

Facts about Putative sodium-coupled neutral amino acid transporter 8.

SLC38A8 3D structure. Source: alphafold

Putative sodium-coupled neutral amino acid transporter 8 (SLC38A8)

Putative sodium-dependent amino acid/proton antiporter. .

Gene Information

Human
Gene Name:	SLC38A8
Uniprot:	A6NNN8
Entrez:	146167

Family

Belongs to:

amino acid/polyamine transporter 2 family

Alternative Names

Putative sodium-coupled neutral amino acid transporter 8

Molecular Weight

Mass (kDA):
46.731 kDA

Genomic Context


Human

Location:	16q23.3
Sequence:	16; NC_000016.10 (84009667..84043372, complement)

Tissue Specificity

Expressed in fetal and adult brain, and spinal cord. In the brain, it is localized in the cell body and axon of the majority of neuronal cells and in a subset of glial cells. Found throughout the neuronal retina, with higher expression levels in the inner and outer plexiform layers and the photoreceptor layer. Very weak expression is also present in the kidneys, thymus, and testes.

Subcellular Localizations

Membrane; Multi-pass membrane protein.

For details, visit:
bosterbio.com/bosterbio-gene-info-cards/SLC38A8

References

PMID: 24290379 by Poulter J.A., et al. Recessive mutations in SLC38A8 cause foveal hypoplasia and optic nerve misrouting without albinism.

PMID: 24045842 by Perez Y., et al. Isolated foveal hypoplasia with secondary nystagmus and low vision is associated with a homozygous SLC38A8 mutation.